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Bharangin, a Diterpenoid Quinonemethide, abolishes constitutive and inducible nuclear factor-kappa B (NF-kappa B) activation by modifying p65 on Cysteine 38 residue and reducing inhibitor of nuclear factor-kappa B alpha Kinase activation, leading to suppression of NF-kappa B-regulated gene expression and sensitization of tumor cells to chemotherapeutic agents

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